Originally posted by wakkawakkawakka
On a slight tangent...had Sweden really reached herd immunity? I know there was a high saturation of confirmed cases...but the mortality rate has been surprisingly low.
I don't think so. I don't think Sweden is suggesting that that is the case. And their mortality rate is somewhat higher than the comparable countries of Norway and Finland.
Originally posted by wakkawakkawakka
On a slight tangent...had Sweden really reached herd immunity? I know there was a high saturation of confirmed cases...but the mortality rate has been surprisingly low.
No. They need to double or triple their infected to reach heard immunity.
Sweden has dropped to #6 on the list of "deaths per million." Other countries are soon to overtake it because they are essentially done with COVID-19.
Originally posted by dadudemonThe concept of herd immunity relies on a disease either not re infecting individuals who previously had it or being less damaging on reinvention. Many viruses, Ebola being the most extreme do neither, we do not know Coronavirus does either... *cringe*
No. They need to double or triple their infected to reach heard immunity.Sweden has dropped to #6 on the list of "deaths per million." Other countries are soon to overtake it because they are essentially done with COVID-19.
Herd immunity for SARS-CoV-2 be much lower, study finds.
Mathematicians from the University of Nottingham and University of Stockholm devised a simple model categorising people into groups reflecting age and social activity level. When differences in age and social activity are incorporated in the model, the herd immunity level reduces from 60% to 43%. The figure of 43% should be interpreted as an illustration rather than an exact value or even a best estimate. The research has been published today in Science.
https://www.sciencedaily.com/releases/2020/06/200623111329.htm
This would match up to what we say in NYC with the serological tests being much closer to herd immunity, if this research checks out.
This is just one piece of good news in a sea of hopeful news.
Stocks did well today, cases appear to be going down (which means the deaths will follow in the coming days as it goes through the cycle), and deaths appear to be dropping in some states.
Unfortunately, some countries are finally hitting their situation in "full swing" and it is worse than ever for them. Japan, Mexico, Brazil, Philippines, to name a few.
https://www.vox.com/2020/7/12/21321653/getting-covid-19-twice-reinfection-antibody-herd-immunity
My patient caught Covid-19 twice. So long to herd immunity hopes?
Emerging cases of Covid-19 reinfection suggest herd immunity could be wishful thinking.
It seems it might be worse second time around too.
Originally posted by BlakemoreHaha Surt posts. The thing is Voltron is the minority here, they argue with different people across the forum. They just as a group are on a lot so their posts are everywhere. I hope Rob hasn't become as bored as Bash did with their ignorance. I find them incredibly funny. Anyway off to work.
http://www.killermovies.com/forums/showthread.php?threadid=669638&pagenumber=25
http://www.killermovies.com/forums/showthread.php?threadid=613830&pagenumber=2357
http://www.killermovies.com/forums/showthread.php?threadid=669317&pagenumber=137Whirly, if you wanna read stupidity, it's hear.
Research suggests coronavirus reinfection is unlikely
But despite the anecdotal reports from doctors about patients becoming reinfected with the coronavirus, researchers say there's no evidence supporting the notion that people can become reinfected with the virus within a short time period."I haven't heard of a case where it's been truly unambiguously demonstrated," said Marc Lipsitch, an epidemiologist at the Harvard T.H. Chan School of Public Health.
For instance, South Korea's Center for Disease Control and Prevention in one study confirmed that, among 285 cases of people who again tested positive for the coronavirus two months after receiving their initial positive test results (including some who were experiencing Covid-19 symptoms two months after their initial diagnoses), none of the patients' new samples contained enough virus particles to allow researchers to grow the virus from the samples in a lab. Researchers said those results indicated that the patients weren't actively infected with the virus, and the diagnostic tests likely had detected dead virus particles that remained in their bodies or generated false-positive positive results.
Further, the researchers noted that none of the patients who re-tested positive for the coronavirus transmitted the pathogen to others.
"It was pretty solid epidemiological and virological evidence that reinfection was not happening, at least in those people," Angela Rasmussen, a virologist at Columbia University, said.
As for studies showing that antibodies for the novel coronavirus decline over time, researchers say that's how antibodies work for a host of viruses.
Michael Mina, an immunologist at Harvard University, said although those studies have left some people "scratching their heads saying, 'What an extraordinarily odd virus that it's not leading to robust immunity,' … they're totally wrong."
"It doesn't get more textbook than this," he added.
And some research has resulted in different findings regarding coronavirus antibody levels over time. For example, results from a separate study that were released preprint and haven't been peer reviewed found that 120 patients who were infected with the coronavirus and experienced mild or moderate symptoms of Covid-19 had stable levels of coronavirus antibodies for at least three months post infection—and in some cases, those levels increased over that time.
In addition, even if someone who had been infected with the coronavirus didn't develop antibodies for the pathogen, a person's T-cells and B-cells could fight off reinfection, public health experts say.
"Even if you don't have a very high level of antibodies, you may be able to respond very rapidly to a challenge and nip it in the bud," said Michel Nussenzweig, head of the laboratory for molecular immunology at Rockefeller University. "You may be able to produce a better … faster response the second time around."
Ultimately, Monica Gandhi, a professor of medicine and associate chief of infectious diseases at the University of California-San Francisco, said, "No one is yet believing in reinfection since there is no good scientific report on it."
So why do some people experience Covid-19 symptoms twice?
So why, then, do some people seemingly recover from Covid-19 but again experience symptoms later?
Clinicians say more research is needed to answer the question of why some patients appear to fall ill with Covid-19 more than once, but some believe that such patients simply relapse because the coronavirus lays dormant in their bodies and reemerges—an occurrence that's been seen with some viruses that often result in lifetime immunity, such as the chickenpox virus, according to Daniel Griffin, an infectious diseases doctor and researcher at Columbia University Medical Center.
It also may be possible that some patients experience a long course of infection that ramps up months after they first contract the coronavirus and initially experience symptoms of Covid-19, some experts have said. And others have speculated that some patients may contract a different virus with similar symptoms to Covid-19 and assume they've been reinfected with the coronavirus.
https://www.advisory.com/daily-briefing/2020/07/24/covid-reinfection
So much for the scare tactics of trying to fearmonger people into believing rampant reinfections of SARS-CoV-2.
Gosh, isn't science great and extremely rare exception cases do not make real science?
A study has revealed that patients who recovered from COVID-19 risk contracting the virus again after six months.
The research findings, the researchers said, has cast further doubt over the prospect of long-lasting immunity from COVID-19.
The team of researchers from the University of Amsterdam had engaged 10 volunteers for 35 years and tested them every month for four seasonal and weaker coronaviruses, which usually cause mild illnesses similar to the common cold.
The truth is nobody knows yet and those dealing in absolutes look very silly.
Interesting pages to follow in regards to covid-19
US
Global
Originally posted by snowdragon
Interesting pages to follow in regards to covid-19US
Global
This one's good too.
Prof Who Founded Anti-Harassment Group Created Hoax Professor Dying Of COVID
"On July 31, BethAnn McLaughlin, the founder of MeTooStem, announced on Twitter that the supposed professor, who went by the name “@Sciencing_Bi” on Twitter, had died of COVID-19 after battling the virus for months. She wrote on Twitter, “She was a fierce protector of people. No one has ever had my back like that,” as Inside Higher Ed noted.
But after numerous Twitter users who communicated with or followed the @Sciencing_Bi account started questioning whether the account was real, McLaughlin finally confessed on Tuesday she had faked the account, with her attorney releasing this statement to The New York Times: “I take full responsibility for my involvement in creating the @sciencing_bi Twitter account. My actions are inexcusable. I apologize without reservation to all the people I hurt.”
LMFAO, oh leftists...
Good news on SARS-CoV-2 immunity: not only is it extremely extremely rare to get sick with it again, the population has somewhere between 20%-40% immunity already from previous coronavirus strains.
Meaning, to hit that oh-so wonderful 60%-70% herd immunity rate is much closer than we think.
SARS-CoV-2 reactive CD4+ T cells have been reported in unexposed individuals, suggesting pre-existing cross-reactive T cell memory in 20-50% of people. However, the source of those T cells has been speculative. Using human blood samples derived before the SARS-CoV-2 virus was discovered in 2019, we mapped 142 T cell epitopes across the SARS-CoV-2 genome to facilitate precise interrogation of the SARS-CoV-2-specific CD4+ T cell repertoire. We demonstrate a range of pre-existing memory CD4+ T cells that are cross-reactive with comparable affinity to SARS-CoV-2 and the common cold coronaviruses HCoV-OC43, HCoV-229E, HCoV-NL63, or HCoV-HKU1. Thus, variegated T cell memory to coronaviruses that cause the common cold may underlie at least some of the extensive heterogeneity observed in COVID-19 disease.
It explains why some get sick, some do not. It also explains why some people who have clearly been heavily exposed still do not test positive - their body can easily wipe out the virus - due to prior immunity.
https://science.sciencemag.org/content/early/2020/08/04/science.abd3871